Published August 1988 | Version v1
Journal article

Mechanism of ductility-loss in neutron-induced metals

  • 1. Kyushu Univ., Fukuoka (Japan). Faculty of Engineering

Description

Irradiation of metals with high-energy particles such as neutron, ion or electron produces points defects (atomic voids, interstitial atoms) and depleted zones where the atomic density is low. The formation of point defect clusters (dislocation loops) causes a decrease in hardening coefficient, leading to a decreased ductility. The life of concentrated gliding (dislocation channelling) in dislocation-loop-free large gliding zones is short, contrary to past predictions, indicating that channelling is not the dominant factor in the decreased hardening coefficient, or decreased ductility. Bud-like secondary gliding occurs to eliminate the elastic strain accompanying the bending of crystals in a large gliding zone while secondary propagation dislocation takes place to complete the bend gliding required to increase plastic deformation, resulting in the cancellation of the dislocation loops in the non-gliding zones. As a result, the resistance to the primary gliding deformation in such zones is greatly reduced to cause a decrease in hardening coefficient, hence a decrease in extension. The decrease in ductility becomes smaller as the strength after neutron irradiation is maintained more strongly by stable obstacles which do not disappear through reaction with dislocation loops. (Nogami, K.)

Availability note (English)

Available from DOI: https://doi.org/10.3327/jaesj.30.663

Abstract (Japanese)

金属は高速中性子で照射されると格子欠陥が導入され、その延性が著しく低下することは、古くから知られている。しかし、この減少は金属の塑性変形機構と関係した複雑なものであるため、未解明のまま現在に至っている。本稿では、この問題に関連した最近の研究に基づいて、この機構を検討し、耐延性低下合金の開発に対する考え方を模索しようとするものである。(著者)

Additional details

Additional titles

Original title (Japanese)
中性子照射金属の延性低下機構

Identifiers

Publishing Information

Journal Title
Nippon Genshiryoku Gakkai-Shi
Journal Volume
30
Journal Issue
8
Journal Page Range
p. 663-670
ISSN
0004-7120
CODEN
NGEGAL

Optional Information

Notes
This record replaces 20029053