Published March 2018 | Version v1
Journal article

Celastrol pretreatment attenuates rat myocardial ischemia/ reperfusion injury by inhibiting high mobility group box 1 protein expression via the PI3K/Akt pathway

  • 1. Division of Cardiovascular Medicine, Department of Medicine, Brigham and Women's Hospital and Harvard Medical School, Boston, MA (United States)
  • 2. Department of Cardiology, Renmin Hospital of Wuhan University, Wuhan, Hubei (China)

Description

Celastrol pretreatment has been shown to protect against myocardial ischemia/reperfusion (I/R) injury, but the underlying mechanism is poorly understood. This study aimed to investigate the cardioprotective effects of celastrol pretreatment on I/R injury and to further explore whether its mechanism of action was associated with the inhibition of high mobility group box 1 protein (HMGB1) expression via the phosphoinositide 3-kinase (PI3K)/Akt pathway.

Availability note (English)

Available from http://dx.doi.org/10.1016/j.bbrc.2018.02.121

Additional details

Identifiers

DOI
10.1016/j.bbrc.2018.02.121;
PII
S0006291X18303528;

Publishing Information

Journal Title
Biochemical and Biophysical Research Communications
Journal Volume
497
Journal Issue
3
Journal Page Range
p. 843-849
ISSN
0006-291X
CODEN
BBRCA9

INIS

Country of Publication
United States
Country of Input or Organization
International Atomic Energy Agency (IAEA)
INIS RN
53054527
Subject category
S60: APPLIED LIFE SCIENCES;
Descriptors DEI
INJURIES; ISCHEMIA; PHOSPHOTRANSFERASES; RATS
Descriptors DEC
ANEMIAS; ANIMALS; CARDIOVASCULAR DISEASES; DISEASES; ENZYMES; HEMIC DISEASES; MAMMALS; ORGANIC COMPOUNDS; PHOSPHORUS-GROUP TRANSFERASES; PROTEINS; RODENTS; SYMPTOMS; TRANSFERASES; VASCULAR DISEASES; VERTEBRATES

Optional Information

Copyright
Copyright (c) 2018 Published by Elsevier Inc.