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Banaszak, J.E.; Argonne National Lab., IL; Reed, D.T.; Rittmann, B.E.
Argonne National Lab., IL (United States). Funding organisation: USDOE Office of Energy Research, Washington, DC (United States)1997
Argonne National Lab., IL (United States). Funding organisation: USDOE Office of Energy Research, Washington, DC (United States)1997
AbstractAbstract
[en] The objective of this work was to investigate the toxicity mechanisms of neptunium and the neptunium-NTA complex towards Chelatobacter heintzii. The results show that metal toxicity of aquo NpO2+ may significantly limit growth of Cl heintzii at free metal ion concentrations greater than ∼ 10-5 M. However, neptunium concentrations ≥ 10-4 M do not cause measurable radiotoxicity effects in C. heintzii when present in the form of a neptunium-NTA complex or colloidal/precipitated neptunium-phosphate. The neptunium-NTA complex, which is stable under aerobic conditions, is destabilized by microbial degradation of NTA. When phosphate was present, degradation of NTA led to the precipitation of a neptunium-phosphate phase
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10 Mar 1997; 41 p; CONTRACT W-31109-ENG-38; Also available from OSTI as DE97007211; NTIS; US Govt. Printing Office Dep
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